Digoxin Ati Medication Template (2024)

FAQs

What are two 2 contraindications for the use of digoxin ATI? ›

Digoxin is contraindicated in the following conditions: Acute myocardial infarction. Hypersensitivity to the drug. Ventricular fibrillation.

As stated, an important prerequisite is to make sure that the apical heart rate is monitored to prevent digoxin toxicity. Remember, digoxin's therapeutic level is between 0.8 and two (2) ng/mL.

You can take digoxin with or without food, but it's best to take it at the same time each day. Most people take it in the morning after breakfast. You'll usually take it once a day. Swallow the tablets whole with a drink of water.

A nurse should assess the apical pulse for a full minute before administering digoxin due to its positive inotropic action (it increases contractility, stroke volume, and, thus, cardiac output), negative chronotropic action (it decreases heart rate), and negative dromotropic action (it decreases electrical conduction ...

Apical pulse should be taken for a full minute before administration of this medication. If the apical pulse is less than 60, the dose should be withheld and the prescribing provider notified. Serum digoxin levels should be monitored, with a normal therapeutic range from 0.8 to 2 ng/mL.

[4] Without the use of digoxin-specific Fab fragments, in a recent review, mortality after digoxin toxicity have been reported to be zero in patients with acute digoxin toxicity and potassium levels less than 5.0 meq/dl, 50% in potassium levels between 5.0 and 5.5, and 100 percent in those with potassium levels above ...

Hold dose and notify health care professional if pulse rate is <60 bpm in an adult, <70 bpm in a child, or <90 bpm in an infant. Notify health care professional promptly of any significant changes in rate, rhythm, or quality of pulse.

It is eliminated primarily through kidneys; therefore, it has a half-life of 36-48 hours in patients who have normal kidney function and 3.5-5 days in patients who are anuric. Therapeutic levels of digoxin are 0.8-2.0 ng/mL.

The electrolyte and renal status of each patient should be ascertained prior to initiating treatment and periodically thereafter. Hypokalemia or hypomagnesemia, for example, may promote the development of digoxin-induced arrhythmias, while impaired renal function may result in higher than anticipated serum drug levels.

What is important to know when taking digoxin? ›

Cautious dosage determination is essential, and lean body weight should be used when determining dosage calculations. Signs of toxicity include nausea, vomiting, visual disturbances, arrhythmia, and weight loss. Discontinue digoxin immediately if signs of toxicity appear.

Gastrointestinal upset is the most common symptom of digoxin toxicity. Patients also may report visual symptoms, which classically present as a yellow-green discoloration, and cardiovascular symptoms, such as palpitations, dyspnea, and syncope.

Digoxin is one of the medications used to manage heart rate, which is considered controlled when the ventricular response is 60 to 80 bpm at rest and 90 to 115 bpm during moderate exercise. First-line therapies for rate control include diltiazem, verapamil, esmolol, and other beta-blockers.

Adult: 250 to 500 micrograms every 4 to 6 hours according to response, up to a total of 1.5mg. Elderly: 125 to 250 micrograms every 4 to 6 hours according to response, up to a total of 500 micrograms.

Nursing implications and nursing considerations include educating patients about potential side effects and interactions with other drugs. Nurses will also need to monitor, or educate the patient to monitor pulse, blood pressure, weight, urine output, electrolyte levels, and digoxin concentrations in the blood.

Digoxin may make your heart beat slower than normal (bradycardia). It may also cause heart rhythm problems that can lead to an irregular heartbeat and fainting. In rare cases, this may cause the heart to suddenly stop beating (cardiac arrest).

Digitalis glycosides are contraindicated in patients with ventricular fibrillation or in patients with a known hypersensitivity to digoxin. A hypersensitivity reaction to other digitalis preparations usually constitutes a contraindication to digoxin.

Quinidine and procainamide are contraindicated. These agents worsen SA, AV, and His-Purkinje conductivity. Additionally, quinidine reduces digoxin tissue binding and renal clearance, thereby increasing digoxin levels.

Because digoxin binds to the K⁺ site of the Na⁺/K⁺-ATPase pump, low serum potassium levels increase the risk of digoxin toxicity. Conversely, hyperkalemia diminishes digoxin's effectiveness.